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Correct mutations and sequences were confirmed by sequencing both strands
Mitochondrial dysfunction leading to oxidative stress also induces FGF21 expression in preclinical disease models (110), while levels of FGF21 are elevated in the circulation and skeletal muscle of patients with myopathies caused by mutations in components of the mitochondrial oxidative phosphorylation machinery (111)
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This was rapidly followed by the upregulation of the PS marks brachyury ( T ), mix paired-like homeobox ( MIXL1 ) and goosecoid ( GSC ) (See Fig
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