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[PubMed] [CrossRef] Cross AJ, Major JM, Sinha R

7 In a survey involving 443 physicians, the reasons for discontinuing GLP-1 RAs were investigated in patients who had stopped treatment within the previous six months

proposed that the GHK tripeptide sequence may arise endogenously as a collagen degradation signal, with GHK-Cu potentially engaging fibroblasts to stimulate new collagen synthesis in response

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By mimicking both GLP-1 and GIP , it works more efficiently to control blood sugar