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In HFD/STZ-induced diabetic mice and HG-treated HK-2 cells, ginsenoside Rg2 prevents the TECs injury by suppressing the proinflammatory NF-B/NLRP3 signaling pathway (171)

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In addition, this sparing effect can also indicate an inhibition of the one-carbon unit-providing action of glycine to the N 510 -methylene-tetrahydrofolate-dependent remethylation of Hcy (Fig