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We showed that cirrhotic (an IGF-1 deficiency condition) rats had diminished amino acid and glucose intestinal absorption [114, 194, 195] and that IGF-1 replacement therapy was able to restore both alterations, suggesting a role of IGF-1 in the position of transporters [196]

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Its clinical data is solid within that specific population: Phase 3 trials demonstrated significant reductions in visceral adipose tissue volume with weekly subcutaneous injection protocols

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