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Importantly, these criteria intentionally exclude peptide systems for which multiple well-designed human studies have demonstrated a lack of efficacy or unacceptable safety profiles
It works by increasing insulin production and lowering glucagon secretion
The bridge from "probiotic evidence in non-GLP-1 populations" to "probiotic use during GLP-1 treatment" rests on biological plausibility and the bidirectional microbiome story described above, not on direct head-to-head trials
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