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These results suggest that reduced homocysteine causes protein thiol homocysteinylation that may induce smooth muscle cell proliferation by binding to a redox receptor and that may exacerbate atherosclerotic lesion formation by inducing collagen expression (Tyagi, 1998)
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Managing Pernicious Anaemia in the time of Corononavirus Dr
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However, these structural features should be interpreted as biochemical clues rather than complete explanations of biological activity
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