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Part B can cover physician-administered injectables that carry FDA approval, such as certain biologics for osteoporosis
In isolated rat aorta preparations, the peptide produced concentration-dependent vasodilation that was endothelium-dependent and abolished by L-NAME, and co-immunoprecipitation work traced the response to phosphorylation of Src, caveolin-1, and endothelial nitric oxide synthase, with reduced binding between caveolin-1 and eNOS [1]
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Adding a peptide with potential hemostatic effects requires more frequent INR monitoring, particularly in patients on warfarin or with baseline coagulopathy
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The approaches work through different mechanisms and tissue concentrations
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