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crucial advancements in AD therapy have been made on the basis of the proposed role of amyloid accumulation in the AD development
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Without a priori knowledge of what insults were applied, and in which order, simply observing the final histological lesion renders it impossible to infer the underlying sequence of events nor the initial trigger
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299 A recessive HNF1A variant associated with young-onset diabetes induces beta cell dysfunction and bias towards alpha cell differentiation I
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