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Pro-inflammatory and proatherogenic mCRP, but not nCRP, induces ROS monocyte/macrophage production and facilitates macrophage uptake of necrotic cells (170, 192) contributing to foam cell formation, atherosclerotic plaque formation and plaque rupture or destabilization (190, 191)
(Associating the action of BPC-157 with VEGFR2 activation and endothelial signaling)
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That means less cell damage and better protection for long-term health
Huang, Y
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