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The protocol is flexible enough to serve both goals
Dimet-Wiley A, Wu Q, Wiley JT, et al
GHK-Cus ability to reduce inflammation and oxidative stress further supports its potential in therapeutic applications for tissue repair
Mechanism of Action Inhibits Nicotinamide N-Methyltransferase (NNMT) Increases intracellular nicotinamide and NAD levels Improves mitochondrial efficiency and energy metabolism Supports lipolysis and fat oxidation Reduces adipocyte (fat cell) growth and proliferation Modulates inflammatory pathways and insulin signaling These effects may lead to enhanced fat metabolism, better blood sugar regulation, improved endurance, and a general increase in metabolic flexibility
Klicek et al., 2013)
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