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The synergy between exogenous MOTS-c and exercise is not merely additive: because MOTS-c mimics one of the molecular consequences of high-intensity exercise (AMPK activation via AICAR accumulation), concurrent exercise training may amplify the peptide's effects on GLUT4 translocation and mitochondrial biogenesis
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aeruginosa in model respiratory cell lines, and other multidrug-resistant Gram-positive and Gram-negative species in vitro (Hijazi et al., 2017
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PPAR agonists as therapeutics for the treatment of Alzheimers disease
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