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Three names sit close together in this space, and they are not interchangeable
For a condition that can cause kidney failure, neurological damage, and cardiovascular disease, that gap is not trivial
Research in cell cultures, rodents, and primates shows delayed aging markers, improved physiological parameters, and increased survival indicators
10.1186/1465-9921-10-101 Respir Res
Several mechanisms have been reported for the increased activity of NRF2 in cancers ( Figure 5 ): (1) somatic mutations in KEAP1 or NRF2 , (2) DNA hypermethylation at the promoter region of KEAP1 , (3) the aberrant accumulation of proteins that disrupt the KEAP1NRF2 interaction, (4) transcriptional up-regulation of NRF2 gene through oncogene-dependent signaling, and (5) the modification of KEAP1 protein through oncometabolites
(GBD 2019 Benign Prostatic Hyperplasia Collaborators) (November 2022)
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