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Indeed, in the present study we found that the relative increases in Ti and Te were substantially different during the peak decreases in fr elicited by fentanyl (approximately + 5 min post-injection, see Fig
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These treatments decreased cell viability and increased both soluble and lipid reactive oxygen species (ROS) generation but did not affect mitochondrial ROS or mitochondrial mass
Certainly, there seems to be a mismatch between the influence of high UA and that of gout in risk of PD, as mentioned above
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