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The proposed mechanism involves beta-3 adrenergic receptor stimulation and enhanced fatty acid oxidation in adipocytes [6]
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doi: 10.1016/j.pbb.2021.173168 28 KurheY.MaheshR
Our data above suggested that a high level of Nrf2 protein in quiescent MuSCs functions to promote MuSC activation instead of ROS suppression
Because the unusual bond formed through the gamma-carboxyl group of glutamate can only be hydrolyzed by one enzyme, gamma-glutamyltranspeptidase, which is present only on the external surfaces of certain cells
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