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A clinical trial registration (NCT02637284) appears on ClinicalTrials.gov with a status listed as unknown since 2016
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408 Spatial metabolomics, built on mass spectrometry imaging, enables high-resolution mapping of metabolites across histological structures, providing insights into spatially regulated metabolic reprogramming that underlies therapeutic resistance
It catalyzes the addition of glycine to -glutamylcysteine created by GCL to form GSH, -L-glutamyl-L-cysteinyl-glycine, a reaction again driven by the hydrolysis of ATP
It acts as a substrate linker for the interaction of Cullin 3 (CUL3)-dependent E3 ubiquitin ligase complex [119], with NRF2 leading to continuous ubiquitination and proteasomal degradation of NRF2 [120], thus representing the main negative regulator of NRF2 activity
This side effect often diminishes over time as the body adjusts to the medication
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