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Its mechanism of action involves mimicking the functions of GLP-1, contributing to blood sugar regulation and weight loss
doi:10.1034/j.1399-5618.2002.01180.x 16
Conclusion In sum, perturbation of Met metabolism may promote the damage process of hepatobiliary injury triggered by viral infections/toxins and/or niche abnormalities, with dysregulated inflammatory and immune responses of the liver, and contribute to the obliteration and eventual fibrosis and atresia of the bile ducts in the initiation/progression of BA
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