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These results indicate that the knockdown of GLS1 affected the metabolic profile of both GBM cell lines, and in U251 cells autophagy was activated, as seen by the increased levels of ATG12 protein after GLS1 knockdown (Fig

Rationale and benefits of trimetazidine by acting on cardiac metabolism in heart failure

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relevant material that could not be included in this review is available at Rights and permissions About this article Cite this article Carmeliet, P., Jain, R

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NF-B plays a critical role in the initiation and perpetuation of inflammation