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Acetylcholine controls GABA-, glutamate-, and glycine-dependent giant depolarizing potentials that govern spontaneous motoneuron activity at the onset of synaptogenesis in the mouse embryonic spinal cord
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These effects were more pronounced in individuals with elevated baseline metabolic biomarkers, such as hyperglycemia and hypo-HDL cholesterol.[14] This suggests that L-carnitine could serve as a valuable adjunct therapy in managing metabolic syndrome and its related risks
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