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S-A-GSH helps to support immune function and to optimize glutathione-dependent hepatic detoxifi cation pathways

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To examine whether AKT signaling is required for leptin-induced Bdnf gene expression, an AKT selective inhibitor, AKTi, was infused into the dentate gyrus to block AKT signaling prior to leptin injection

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Gem and HU did not strongly activate p53 and/or induce p21 expression in HT-1080 Control, p53 KO or p21 KO cells (Fig 4C)

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