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The risk of hypoglycemia after an injection is related to the duration of action of the insulin [see Clinical Pharmacology (12.2)] and, in general, is highest when the glucose lowering effect of the insulin is maximal
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GLP-1RAs suppress hyperglycemia-induced proliferation, migration and apoptosis of VSMCs via suppressing the ERK1/2 and PI3K/Akt pathways [10]
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