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Its appetite-suppressing effects stem from multiple proposed physiological mechanisms that target both central and peripheral pathways involved in hunger regulation

and that "relative enrichment of the ASD risk SNPs from the respective evolutionary cluster or biological interaction networks may help in addressing the phenotypic diversity in ASD", with "cognitive genomic tradeoff signatures impacting the biological networks [explaining] the paradoxical phenotypes in ASD"

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The result is a significant boost in energy levels and overall well-being