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Because unaddressed B12 deficiency can lead to irreversible neurological damage, early detection and accessible intervention are essential
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indeed, an SLC1A5-deficient mouse model shows decreased induction of T helper 1 (Th1) and Th17 cells but not Th2, regulatory T (Treg), or CD8(+) T cells, leading to a decrease in proinflammatory T-cell responses 134
Yes, that's the historical origin
It also comes down to how much research you want behind your regimen
It has been suggested that riboflavin therapy increases miR-203 expression, which inhibits c-Jun expression, thus increasing neuronal survival (38)
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