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MRC (UK)

We will continue to illuminate this largely uncharted area in future work.

In posterior segments of the gut (e.g., the cecum and proximal large intestine), gut microbes directly cleave the CN bond of carnitine to release TMA, which subsequently enters the liver through the portal circulation, is converted to TMAO by hepatic flavin monooxygenases (FMOs) [242, 243], and eventually enters the blood circulation [244]

With Fe2+, the enzyme is then able to continue to hydroxylate proline and lysine

Figure 1 presents a flowchart of the study selection process

At the translational clinical level, L-carnitine primarily targets hepatic steatosisthe earliest pathological step in MASLD