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Research has shown that Epithalon can influence oxidative stress and cell repair

Tanaka et al

Using a well-established GEMM of LUAD ( Kras LSL-G12D/+ , Tp53 fl/fl , Rosa26 LSL-Cas9/LSL-Cas9 ), we initiated Kras G12D/+ , Tp53 /- (KP) tumors by intratracheal delivery of lentiviruses expressing CRE-recombinase and dual sgRNAs 38 against either Gpx4 (sg Gpx4 ) or non-targeting control (sg Neo ), enabling CRISPR/Cas9-mediated Gpx4-knockout (Gpx4 KO ) or wildtype (WT) control tumors, respectively

IGF-DES activates these same pathways via IGF-1R but without the prior IGFBP buffering that attenuates native IGF-1 signalling

Unchecked free radicals continuously damage cellular lipids and proteins, triggering a vicious cycle of systemic inflammation

Potent Anti-Inflammatory and Vascular Effects BPC-157 promotes nitric oxide synthesis and reduces pro-inflammatory cytokines (IL-6, TNF-)