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Supplementary information List of consortium members

Data availability The data that support the findings of this study are openly available in TCGA database of PTCs utilizing the Firehouse web browser from the Broad Institute ( using TIMER2.0 ( a computational software for analyses of tumor immunologic, clinical, and genomic features 35

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281 Importantly, while acute or overwhelming endoplasmic reticulum stress can induce cell death, chronic UPR activation favors drug tolerance and persistence, positioning endoplasmic reticulum stress adaptation as a systems-level resistance mechanism complementary to oncogenic signaling network adaptation

5 For instance, Rab31 promotes EMT and cisplatin resistance in stomach adenocarcinoma via the Stat3/MUC-1/Twist1 axis