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When cyp2e1 knockout mice that lack the ability to produce the CYP2E1 enzyme were challenged with acetaminophen, they were found to be considerably less sensitive to its hepatotoxic effects than wild-type animals, indicating that this CYP is the principal enzyme responsible for the metabolic conversion of the drug acetaminophen to its active hepatotoxic metabolite NAPQI (Figure 2)

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In fact, up to 15% of people have been shown, due to their genetics, to have a problem with this conversion process (12)

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This protein is also available as GMP compliant Cell Therapy Grade, to enquire email [email protected]

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