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Results Obese rats exhibit marked hyperphagia accompanied by suppression of GLP-1 in NTS To evaluate the successful establishment of the obesity model and further assess central GLP-1 expression levels in obese rats, we monitored phenotypic changes in these animals during continued administration of different diets post-model induction
Low thyroid function can drive weight gain and fatigue
However, mean WL with GLP-1 RAs remains substantially lower than that achieved with BS and treatment response to GLP-1 RAs is heterogeneous
However, unlike GIPR, GLP-1R are present in several other tissues such as peripheral nervous system, lung and heart (Figure 2) [19]
