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This supports the hypothesis about the hemolysis-preventing role of eryptosis, since detrimental internal or external perturbations triggering eryptosis might continue to act, overcoming the adaptive capacity of cells, causing lysis

Im not saying that to be dramatic

For the systemic injury and recovery uses that TB-500 is actually marketed for, the supporting data are animal models and in-vitro work on thymosin beta-4 and its actin-binding domain

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But I am clear that mental health claims are speculative, and choosing it specifically for psychiatric benefit is not evidence-based