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Civinini, R

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However, whether LA could exert the protective role in FA-induced AKI, its dosage and underlying mechanism remained unknown

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Understanding these mechanistic divergences allows principal investigators to select the precise tool compound required for their specific neurobiological hypothesiswhether studying acute signaling cascades, neuroinflammation, or long-term structural plasticity

Therefore, the study of melatonin in the context of depression must acknowledge its regulatory effect via chronobiotic (endocrine), paracrine and autocrine functions