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Metabolism 52 , 699704 (2003)

Stimulation of extracellular matrix synthesis The most reproducible cell-level finding is that GHK-Cu stimulates dermal fibroblasts to produce more extracellular matrix

The two compounds operate through complementary but mechanistically distinct pathways

TS inhibition by 5FU regulates mitochondrial biogenesis through mTORPGC1 signalling 5FU has a multimodal mechanism of action: promoting DNA damage directly via its metabolite FdUTP or by inhibition of TS via FdUMP, depleting the pool of thymidine available for DNA synthesis, and causing RNA damage via FUTP (Fig

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Tripathi R, Singh B, Bisht S, Pandey J (2009) L-ascorbic acid in organic synthesis: an overview