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Oligodendrocytes are notably susceptible to hypoxia [278], and the oxidative stress resulting from chronic hypoxia is a critical mechanism in neurodegeneration and axonal loss in multiple sclerosis

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Found in plasma, saliva, and urine, it has become a central focus of laboratory studies thanks to its ability to interact with biological pathways linked to skin repair, tissue regeneration, antioxidant protection, and cellular signaling

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