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Other reported mechanisms include activation of FAK-paxillin complexes (which help cells migrate and attach to surfaces during wound healing), JAK-2 signaling, and ERK1/2 pathway activity (Sikiric et al., Current Neuropharmacology, 2016

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Originally uncovered during the exploration of organic anion transporter-like (OAT-like) molecules in the gene database, URAT1s functional aspects were revealed through expression studies in African clawed frog oocytes [56]