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Epidemiology Eight patients have been reported worldwide

Lypopolysaccharide stimulation may lead to HMGB1 to export from the nucleus to cytoplasm where it condenses into secretory lysosomes and is actively secreted from monocytes and macrophages ( in vitro studies on primary peritoneal macrophages from Gstp / mice and wild-type mice, and cell lines of RAW264.7 and THP-1 indicated that GSTP inhibited LPS-induced HMGB1 release through preventing its translocation from the nucleus to cytoplasm

Despite adequate oxygenation and circulation, excitatory neurotransmitters and free radicals continue to be released, phosphorus reserves are depleted, inflammatory factors are involved, and brain injury is substantial