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Experimental studies conducted in animals over the past two decades consistently demonstrate that disruptions in the light-dark schedule induce inflammatory responses across a range of biological systems, with several findings suggesting potential relevance to BC

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2.5.5 Insights from in vitro cell-based models A range of in vitro models has been employed to delineate the cellular basis of imatinib-induced reproductive toxicity as depicted in Table 6

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BPC-157 complements these mechanisms through its activation of VEGFR2 and eNOS pathways that promote angiogenesis and improve endothelial function, with the peptide's nitric oxide generation supporting vasodilation and reducing thrombotic risk while its growth factor sensitization enhances vascular responses to ischemic conditions