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A., Karas, V
Summary Together, these findings demonstrate that elevated glucose results in actin polymerization that impedes both phases of biphasic insulin secretion, and that GLP-1 can restore glucose sensitivity and promote insulin release by correcting errors in actin dynamics at both phases
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Paradoxically, hyperestrogenism was reported to cause a state of IR that could share in the development of gestational diabetes and PCO [49]
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