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Moreover, advanced light-dark schedules in genetically predisposed mice (e.g., K-ras/p53 mutants) led to enhanced tumor growth and cell proliferation with increased oncogene c-Myc expression, while clock gene knockouts have tumor suppressive effects 203

This supports the idea that GLP-1 RAs as a class are likely friends rather than foes to erectile function, insofar as they ameliorate the underlying causes of ED

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