Understanding the functioning of the antioxidant system in eukaryotic cells is important for the assessment of factors that may affect the pro- and antioxidant balance
Entre las alteraciones que se producen en el metabolismo cardiaco durante la fase isqumica del infarto de miocardio (IM) estn la privacin de oxgeno, de nutrientes y de factores de supervivencia, y la acumulacin de residuos en los cardiomiocitos, provocando la activacin de procesos de muerte celular y dando lugar al aturdimiento e hibernacin del miocardio y, finalmente, a la afectacin de la funcin contrctil1,2
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Notably, total IGF-I and IGFBPs were unchanged over this short-duration window, dissociating the observed bone-growth effect from sustained IGF-1 axis activation in this model
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