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In yeast and plants, we observed almost no NADPH consumption upon oxidative challenge with H 2 O 2 or diamide in the absence of a cytosolic glutathione reductase (Figs
Cambronne, X
Our findings demonstrate that knockout of palmitoyltransferase DHHC9 or disruption of DHHC9-mediated GLUT1 S-palmitoylation by site-specific point mutation inhibited growth and colony formation of GBM cells in vitro and GBM tumorigenesis in vivo
bovis BCG infections, alongside antibiotics
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