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Potential targets of microglia in the treatment of neurodegenerative diseases: mechanism and therapeutic implications
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GSH and cathecol estrogens
3.3 Modulating CAFs and ECM CAFs contribute to immunotherapy resistance through multiple mechanisms, including physical barrier formation, secretion of immunosuppressive cytokines, and metabolic reprogramming of the TME (34)
Hormonal fluctuations are another key consideration: Rapid weight loss can significantly alter SHBG and free testosterone levels, potentially leading to symptoms of androgen excess or deficiency even when the prescribed testosterone dose remains unchanged
