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3.2 Therapeutic potential in obesity Although the incretin effects of GIPR undoubtedly play a crucial role in preventing postprandial glucose excursion, there is still intensive debate on whether the GIPR should be activated or inhibited for the treatment of obesity
Even in severe VHT, the prognosis seems to be improved if VPA therapy is promptly discontinued [62]
PPAR is expressed in many glucoregulatory organs 12,13,14,15,16,17 , where its activation improves hepatic lipid and cholesterol metabolism while decreasing liver fibrosis and expression of proinflammatory cytokines 18
Furthermore, GLP-1 reduces the inactivating phosphorylation of PDH, effectively redirecting glucose metabolism away from anaerobic glycolysis toward more efficient mitochondrial oxidation (Torres et al., 2016)
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