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This suggests that changes in the incretin system during colitis may influence blood glucose levels and may act as a prospective novel therapeutic target for CD [225]

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GBM cells increase conversion of tryptophan to kynurenine via upregulation of indoleamine 2,3-dioxygenase (IDO1/IDO2) and tryptophan 2,3-dioxygenase (TDO2), depleting local levels of tryptophan and impairing T-cell functioning (107, 108)