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Mechanistically, OA suppresses pro-inflammatory cytokines (e.g., IL-17A, TNF-) by inhibiting nuclear factor kappa-B (NF-B) and signal transducer and activator of transcription 3 (STAT3) signaling pathways [8,9,10], attenuates oxidative stress via nuclear factor erythroid 2-related factor 2 (Nrf2)/antioxidant response element (ARE) axis activation [11, 12], and restores immune homeostasis by modulating Th17/Treg balance [9, 13]

Jones, et al

It is primarily studied for its ability to upregulate telomerase activity, the enzyme responsible for rebuilding telomeres (the protective caps at the ends of chromosomes)

Oral sulforaphane increases Phase II antioxidant enzymes in the human upper airway

Bottom line: Without trial data, every dose is an experiment and the safety implications are unknown FAQ Whats the Maximum Reported Dose People Use

Vercellini P, Buggio L, Berlanda N, Barbara G, Somigliana E, Bosari S