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Hernandez-Lopez, R
The integrity of the endothelium and oxidative damage could be restored and reversed respectively, which was linked to the increased expression and internalisation of VEGFR2
Research in cell culture and rodent models consistently demonstrates that MOTS-c accelerates this translocation independently of insulin, offering a distinct and additive mechanism to insulin sensitizers like Metformin [1]
The peptide also attracts immune cells to wound sites, stimulates nerve growth, and promotes angiogenesis
Parkinson's disease: an overview of current treatment options
Robbins, J., et al
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