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Figure 4 Alterations in the intracellular redox status are often associated with GSH depletion (Polonikov, 2020) and endogenous GSH deficiency, due either to decreased biosynthesis and/or increased consumption, is a marked contributor to the pathogenesis of numerous diseases via mechanisms including oxidative stress and inflammation
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Conversely, the protein SKCRP14.1 promoted PCD in response to antimonials but conferred protection against miltefosine-induced PCD ( 4.2 Complex mechanisms of miltefosine resistance in Leishmania Recent studies have identified multiple mechanisms underlying miltefosine (MF) resistance in Leishmania species, posing significant challenges to effective treatment
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Future studies are warranted to explore the molecular mechanisms involved in the ALAs protective effects against prenatal cytarabine-induced hepatotoxicity
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