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In turn, these compromised mitochondria produce excess ROS, which further stimulates pro-inflammatory gene expression through MAPK and NF-B signaling, creating a self-reinforcing feedback loop that amplifies the inflammatory cytokines response
Zalachoras I, et al
Endpoints The primary endpoint was glycaemic control (change in HbA1c) as a measure of the impact of the addition of liraglutide 1.8 mg once daily on the clinical efficacy of LAGB in treating T2DM between randomisation and 6 months
Use carries potential risks including changes to moles and nevi that require dermatological monitoring
The clinical significance of this slight difference is debatable, but the effect is mentioned here as a possible off-target effect of DPP-4 inhibitors
The accuracy is evaluated on the basis of the difference between the one minute average value and the required set point of the active power
