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When these two lines cross around age 45, cells become increasingly vulnerable to damage they can no longer adequately defend against
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However, emerging evidence indicated that ferroptosis is an autophagic cell death process 48,49,50
The silencing of the DNA methylation of the elongation of very long-chain fatty acid protein 5 (ELOVL5) and fatty acid desaturase 1 (FADS1) leads to ferroptosis resistance, and these two enzymes are usually upregulated in mesenchymal-type gastric cancer cells (Lee J.-Y
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