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Supports analytical quantification in preclinical biological matrices

Parkinson and other models -synuclein A53T or A30P : Parkinson models with Lewy-body-like pathology

Tramutola A, Triplett JC, Di Domenico F, Niedowicz DM, Murphy MP, Coccia R, Perluigi M, Butterfield DA (2015) Alteration of mTOR signaling occurs early in the progression of Alzheimer disease (AD): analysis of brain from subjects with pre-clinical AD, amnestic mild cognitive impairment and late-stage AD

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Nevertheless, pharmacological maintenance or restoration of GSH levels can inhibit NF-B signaling and cytokine release, thereby preventing excessive or chronic inflammation.37 Thus, although the present IHC findings suggest possible modulation of NF-Brelated pathways, definitive mechanistic interpretation requires molecular-level confirmation through gene or protein expression analyses