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The LR3 modification, which involves the substitution of an arginine for glutamic acid at position 3 and a 13-amino-acid extension at the N-terminus, is believed to support its affinity for IGF receptors while reducing its interactions with IGF-binding proteins

Cellular senescence or stemness: hypoxia flips the coin

The binding of NLR to ligands induces the activation of downstream NF-B or MAPK pathways and subsequently leads to the secretion of pro-inflammatory cytokines [29]

2001 Mar;3(2):181-188

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