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Being the central hub for key pathways and signaling cascades, ER is considered to be requisite for cell fate and survival, while any dysfunction in these processes results in ER stress [293, 294], which can be used to initiate cancer cell death through apoptosis by promoting the cleavage of caspase 8 or by activating anti-tumor immunity by enhancing the expression of immunogenic proteins on the surface of cancer cells [295, 296]

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Clinical utility of cardiovascular risk scores for identification of people with type 2 diabetes more likely to benefit from either GLP-1 receptor agonist or SGLT2 inhibitor therapy

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Selenite is a substrate for calf thymus thioredoxin reductase and thioredoxin and elicits a large non-stoichiometric oxidation of NADPH in the presence of oxygen