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1) Pro-angiogenic signaling (VEGFR2 Akt eNOS) In cell and animal models, BPC-157 increases VEGFR2 expression/internalization and activates downstream Akt-eNOS signaling, supporting endothelial tube formation and blood-flow recovery after ischemia

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investigated the effects of tamoxifen on the respiratory rate and mitochondrial phosphorylation efficiency in rats, showing that tamoxifen reduces the production of adenosine triphosphate and even leads to cell death in liver cells 24

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